Steroid induced glaucoma causes

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Optic disc photographs may be taken to assess the degree of damage - if any -  to the optic nerve, plus a visual-field test to determine the range of peripheral vision. Advice may be given to gradually discontinue topical medication in one eye to see if the pressure improves. Reduce the concentration or dosage of the steroid. Change to a less potent steroid such as FML (fluorometholone), Vexol (rimexalone) or Lotemax (loteprednole). Change to an oral nonsteroidal anti-inflammatory drug, eg. ibuprofen. Treat as for open-angle glaucoma. Where inflammation is moderate to severe, steroids are usually increased initially to reduce the inlammation using a topical hypotensive agent such as a prostaglandin analogue or a beta-blocker.

A familiarity with steroid-induced glaucoma or ocular hypertension is essential to the care of a uveitic patient. Gonioscopy is required to define the open- or closedangle mechanism for elevated IOP. Careful charting will help the clinician to determine if the increase in IOP relates to the inflammation or the steroid therapy. Steroid-sparing therapy or the avoidance of steroids in affected or at-risk patients can help avoid or treat a steroid response. Comanagement with a rheumatologist or uveitis specialist is likely required (Figure 2).

Long term use of topical & systemic steroids produce secondary open angle glaucoma similar to chronic simple glaucoma. The increased IOP caused by prolonged steroid therapy is reversible but the damage produced by it is irreversible. In this study, we analysed 25 patients (44 eyes) with steroid induced glaucoma, who reported to us with dimness of vision, haloes and elevated . and were using steroids for long duration due to various causes. The behaviour of the . due to different steroid preparations, the type of lenticular change, and the management of those cases are discussed in this paper. From our study we conclude that dexamethasone and betamethasone both topical as well as systemic are more potent in producing glaucoma and cataract than medrysone and prednisolone. The condition is reversible without permanent damage when the duration of steroid therapy is short and vice versa.

Steroid induced glaucoma causes

steroid induced glaucoma causes

Long term use of topical & systemic steroids produce secondary open angle glaucoma similar to chronic simple glaucoma. The increased IOP caused by prolonged steroid therapy is reversible but the damage produced by it is irreversible. In this study, we analysed 25 patients (44 eyes) with steroid induced glaucoma, who reported to us with dimness of vision, haloes and elevated . and were using steroids for long duration due to various causes. The behaviour of the . due to different steroid preparations, the type of lenticular change, and the management of those cases are discussed in this paper. From our study we conclude that dexamethasone and betamethasone both topical as well as systemic are more potent in producing glaucoma and cataract than medrysone and prednisolone. The condition is reversible without permanent damage when the duration of steroid therapy is short and vice versa.

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